The Shaken Baby Syndrome Myth
renamed "Abusive Head Trauma" or "Non-Accidental Injury"



* SBS began as an unproven theory and medical opinions, now discredited by biomechanical engineering studies
* No DIFFERENTIAL DIAGNOSIS done to eliminate other causes, abuse assumed without evidence
* Shaken Baby diagnostic symptoms not caused by shaking
* Child protective agencies snatch children, destroy families based on medical accusations without proof of wrong-doing
*Poor or deceptive police investigations, falsified reports, perjured testimony threaten legal rights, due process
* Prosecutors seek "victory", over justice; defense attorneys guilty of ineffective counsel, ignorance, lack of effort
* Care-takers threatened, manipulated, in order to force plea bargains, false confessions
* A fractured criminal justice system--a big piece for the rich, a small piece for the poor, and none for alleged SBS cases.



Related websites/ important people and projects ShakenBabySyndrome/Vaccines/YurkoProject
"Shaken Baby Syndrome or Vaccine Induced Encephalitis-- Are Parents Being Falsely Accused?" by Dr Harold Buttram, with Christina England (WEBSITE)
Evidence Based Medicine and Social Investigation:
EBMSI conferences, resources and information Articles and Reports
VacTruth: Jeffry Aufderheide; The SBS conection and other dangerous or deadly side effects of vaccination true, suppressed history of the smallpox vaccine fraud and other books:
Patrick Jordan
Sue Luttner, must-read articles and information on Shaken Baby Syndrome: her resources link
The Amanda Truth Project: Amanda's mother speaks out at symposium
Tonya Sadowsky



Casey Laverty was born on Nov.20th, 2000. Her mother hemorrhaged for 2 days after giving birth, prior to which she lived on a diet of small amounts of junk food, coffee, excessive sugar, no pre-natal vitamins, and she smoked before, during and after giving birth. Casey had abnormally swollen cheeks and a rolly-poly appearance in spite of a 10lb weight gain by her mother during the pregnancy. The mother, Melissa was unable to breast feed and pumped milk in scanty amounts. According to John, the father, someone came in to vaccinate the baby the next day (21st) with Hep B (according to the records) but he heard them say "triple vax". He refused the shot due to known adverse reactions, but was falsely told, "You have no choice, it's the law." No warning on possible reactions was provided, as required by law. Two days later, according to the mother, on Thanksgiving Day ("corrected" pediatric records claim it was on the 24th), Casey had been lethargic and wouldn't take her bottle when she gave a cry. John picked her up and her eyes rolled back in her head and she seemed to stop breathing temporarily. He noted a yellow tint to her skin and bluishing tinge around her mouth. She didn't respond to voice or finger-snapping near her face, so the parents took her to the E.R. at Mary Bridge Hospital.




Sepsis following vaccination   Antibiotic Cefotaxime Side Effects   Ampicillin Side Effects

Antibiotic Ceftriaxone: Adverse Effects "Ceftriaxone for Injection and Dextrose Injection should not be administered to hyperbilirubinemic neonates, especially prematures."

*Dr. Hurt described the heart rate as normal. Dr. Tart described "cardiac anomalies/abnormalities", possible apnea, possible reflux
*Jaundice related to BREAST FEEDING?

* Dr. Tart claimed this was the first child for both parents. In fact, this was John's second child. He had an older daughter, born in July, 1999
* He said both parents were healthy, but in fact the mother had a number of health problems including abnormal bleeding at delivery.
*Heart rate said to be "normal" although fluctuations were documented.
* He claimed there was no history in the family of respiratory problems, but in fact John's older daughter had severe respiratory problems soon after birth, with rales in the lungs, until her diet was changed to accommodate diagnosed food intolerances, and both John and his brother were very sick in infancy until taken off cow's milk and given goat's milk. John was unaware of the respiratory problems his older daughter had because she was in the care of her maternal grandmother in another part of the state at that time.


* Dr. Clapper quoted previous errors, such as this was the first child for both parents when John had another child.
*"No complications of pregnancy or delivery"--the complications were listed previously.
*"It was unsure whether the monitor was intact during the lower readings" --how would the monitor be defective at lower readings but accurate at normal readings?
*"Uncertain who their primary physician was" --they were not assigned a primary physician at the prenatal clinic the parents attended.

Sepsis refers to bacterial infection of the bloodstream. It means that the normal defenses of the body against overwhelming infection are breaking down and bacterial germs are loose and multiplying in the bloodstream. Once this state has arisen, the patient&/39;s chances for recovery begin to dwindle rapidly. Sepsis is a medical emergency requiring swift and decisive intervention. Septic shock and death will follow if this condition is left untreated for long.

For this reason, that the consequences of missing sepsis are so dire, and because the early signs of sepsis may be quite subtle, physicians often perform what is referred to as the "septic workup," which means obtaining culture specimens of blood, urine, and spinal fluid (via lumbar puncture or spinal tap). While awaiting these test results, powerful antibiotics are administered intravenously in high doses until the situation is more clear - whether the patient is truly gravely infected or perhaps suffering from another illness, often viral, which is not serious.

Most septic workups are performed on very young infants, because these children in particular are the most difficult to assess on clinical grounds (physical examination, history or presence of fever, and routine lab tests such as blood count). Most septic workups are false alarms, but they are a small price to pay for saving young lives.


Sick children

The grunting of sick persons is different from the occasional grunt of the happy, healthy child. Grunting caused by illness is more regular, often with every breath. The child usually appears to be in some distress or to be ill. Grunting respirations in a baby with a fever or who appears to be sick is a very bad sign and demands immediate emergency medical attention. The grunting respirations could represent

* pneumonia
* asthma or in a baby, bronchiolitis with significant oxygen lack - look out for a blue coloration around the lips - cyanosis
* sepsis
* meningitis
* heart failure with flooding of the lungs with fluid, in a child either known to have heart disease or previously undiagnosed

Severe allergic reactions (rash; hives; itching; difficulty breathing; tightness in the chest; swelling of the mouth, face, lips, or tongue); bloody or severe diarrhea; chest pain; chills; fever; severe nausea or vomiting; stomach pain or cramps; unusual bruising or bleeding; vaginal irritation or discharge; vein inflammation or tenderness; white patches in mouth.

Liver– A moderate rise in serum glutamic oxaloacetic transaminase (SGOT) has been noted, particularly in infants, but the significance of this finding is unknown. Mild transitory SGOT elevations have been observed in individuals receiving larger (two to four times) than usual and oft-repeated intramuscular injections. Evidence indicates that glutamic oxaloacetic transaminase (GOT) is released at the site of intramuscular injection of Ampicillin sodium and that the presence of increased amounts of this enzyme in the blood does not necessarily indicate liver involvement.

Hemic and Lymphatic Systems– Anemia, thrombocytopenia, thrombocytopenic purpura, eosinophilia, leukopenia, and agranulocytosis have been reported during therapy with the penicillins. These reactions are usually reversible on discontinuation of therapy and are believed to be hypersensitivity phenomena.

Hepatic: Increased AST (SGOT), ALT (SGPT), alkaline phosphatase, and LDH.

Hematologic: Decreased hemoglobin, hematocrit, RBC, WBC, neutrophils, lymphocytes, platelets and increased lymphocytes, monocytes, basophils, eosinophils, and platelets.

Blood Chemistry: Decreased serum albumin and total proteins.

Renal: Increased BUN and creatinine.

Urinalysis: Presence of RBC's and hyaline casts in urine.

The following adverse reactions have been reported with ampicillin-class antibiotics and can also occur with Ampicillin and Sulbactam for Injection.


Gastritis, stomatitis, black "hairy" tongue and enterocolitis. Onset of pseudomembranous colitis symptoms may occur during or after antibiotic treatment.

Hypersensitivity Reactions

Urticaria, erythema multiforme, and an occasional case of exfoliative dermatitis have been reported. These reactions may be controlled with antihistamines and, if necessary, systemic corticosteroids. Whenever such reactions occur, the drug should be discontinued, unless the opinion of the physician dictates otherwise. Serious and occasional fatal hypersensitivity (anaphylactic) reactions can occur with a penicillin.


In addition to the adverse laboratory changes listed above for Ampicillin and Sulbactam, agranulocytosis has been reported during therapy with penicillins. All of these reactions are usually reversible on discontinuation of therapy and are believed to be hypersensitivity phenomena. Some individuals have developed positive direct Coombs Tests during treatment with Ampicillin and Sulbactam, as with other beta-lactam antibiotics.

Summary of Interactions for Ampicillin
Depletion or interference
   Vitamin C*
   Vitamin K*
Side effect reduction/prevention
Bifidobacterium longum*
Lactobacillus acidophilus*
Lactobacillus casei*
Saccharomyces boulardii*
Saccharomyces cerevisiae*
Vitamin K*

Interactions with Supplements

Vitamin C

Test tube studies show that ampicillin significantly reduces the amount of vitamin C in the blood.1 Controlled research is needed to determine whether individuals might benefit from supplementing vitamin C while taking ampicillin.


A common side effect of antibiotics is diarrhea, which may be caused by the elimination of beneficial bacteria normally found in the colon. Controlled studies have shown that taking probiotic microorganisms-such as Lactobacillus casei, Lactobacillus acidophilus, Bifidobacterium longum, or Saccharomyces boulardii-helps prevent antibiotic-induced diarrhea.2

The diarrhea experienced by some people who take antibiotics also might be due to an overgrowth of the bacterium Clostridium difficile, which causes a disease known as pseudomembranous colitis. Controlled studies have shown that supplementation with harmless yeast-such as Saccharomyces boulardii3 or Saccharomyces cerevisiae (baker's or brewer's yeast)4-helps prevent recurrence of this infection. In one study, taking 500 mg of Saccharomyces boulardii twice daily enhanced the effectiveness of the antibiotic vancomycin in preventing recurrent clostridium infection.5 Therefore, people taking antibiotics who later develop diarrhea might benefit from supplementing with saccharomyces organisms.

Treatment with antibiotics also commonly leads to an overgrowth of yeast (Candida albicans) in the vagina (candida vaginitis) and the intestines (sometimes referred to as "dysbiosis"). Controlled studies have shown that Lactobacillus acidophilus might prevent candida vaginitis.6

Vitamin K

Several cases of excessive bleeding have been reported in people who take antibiotics.7, 8, 9, 10 This side effect may be the result of reduced vitamin K activity and/or reduced vitamin K production by bacteria in the colon. One study showed that people who had taken broad-spectrum antibiotics had lower liver concentrations of vitamin K2 (menaquinone), though vitamin K1 (phylloquinone) levels remained normal.11 Several antibiotics appear to exert a strong effect on vitamin K activity, while others may not have any effect. Therefore, one should refer to a specific antibiotic for information on whether it interacts with vitamin K. Doctors of natural medicine sometimes recommend vitamin K supplementation to people taking antibiotics. Additional research is needed to determine whether the amount of vitamin K1 found in some multivitamins is sufficient to prevent antibiotic-induced bleeding. Moreover, most multivitamins do not contain vitamin K.
Interactions with Herbs


1. Alabi ZO, Thomas KD, Ogunbona O, Elegbe IA. The effect of antibacterial agents on plasma vitamin C levels. Afr J Med Med 1994;23:143-6.
2. Elmer GW, Surawicz CM, McFarland LV. Biotherapeutic agents. A neglected modality for the treatment and prevention of selected intestinal and vaginal infections. JAMA 1996;275:870-6 [review].
3. Elmer GW, Surawicz CM, McFarland LV. Biotherapeutic agents. A neglected modality for the treatment and prevention of selected intestinal and vaginal infections. JAMA 1996;275:870-6 [review].
4. Schellenberg D, Bonington A, Champion CM, et al. Treatment of Clostridium difficile diarrhoea with brewer's yeast. Lancet 1994;343:171-2.
5. Surawicz CM, Elmer GW, Speelman P, et al. Prevention of antibiotic-associated diarrhea by Saccharomyces boulardii: A prospective study. Gastroenterol 1989;96:981-8.
6. Elmer GW, Surawicz CM, McFarland LV. Biotherapeutic agents. A neglected modality for the treatment and prevention of selected intestinal and vaginal infections. JAMA 1996;275:870-6 [review].
7. Suzuki K, Fukushima T, Meguro K, et al. Intracranial hemorrhage in an infant owing to vitamin K deficiency despite prophylaxis. Childs Nerv Syst 1999;15:292-4.
8. Huilgol VR, Markus SL, Vakil NB. Antibiotic-induced iatrogenic hemobilia. Am J Gastroenterol 1997;92:706-7.
9. Bandrowsky T, Vorono AA, Borris TJ, Marcantoni HW. Amoxicllin-related postextraction bleeding in an anticoagulated patient with tranexamic acid rinses. Oral Surg Oral Med Oral Pathol Oral Radiol Endod 1996;82:610-2.
10. Kaiser CW, McAuliffe JD, Barth RJ, Lynch JA. Hypoprothrombinemia and hemorrhage in a surgical patient treated with cefotetan. Arch Surg 1991;126:524-5.
11. Conly J, Stein K. Reduction of vitamin K2 concentration in human liver associated with the use of broad spectrum antimicrobials. Clin Invest Med 1994;17:531-9.
12. Attel OA, Ali AA, Ali HM. Effect of khat chewing on the bioavailability of ampicillin and amoxicillin. J Antimicrob Chemother 1997;39:523-5.
13. Hamid S, Beg AE. Influence of ethnic diets on ampicillin bioavailability and pharmacokinetics in healthy Pakistani subjects. Pol J Pharmacol Pharm 1987;39:337-42.
14. Rao SS, Edwards CA, Austen CJ, et al. Impaired colonic fermentation of carbohydrate after ampicillin. Gastroenterology 1988;94:928-32.
15. Lutz M, Espinoza J, Arancibia A. Effect of structured dietary fiber on bioavailability of amoxicillin. Clin Pharmacol Ther 1987;42:220-4.
16. Nunez-Vergara LJ, Yudelevich J, Squella JA, Speisky H. Drug-acetaldehyde interactions during ethanol metabolism in vitro. Alcohol Alcohol 1991;26:139-46.

Cefotaxime Adverse Reactions

Caution with hypersensitivity to penicillin; adjust dosage in patients with renal impairment; may cause neutropenia, thrombocytopenia, eosinophilia, positive Coombs test, and elevated BUN, creatinine, and liver enzymes

Local (4.3%): Injection site inflammation with IV administration. Pain, induration, and tenderness after IM injection.

Hypersensitivity (2.4%): Rash, pruritus, fever, eosinophilia and less frequently urticaria and anaphylaxis.

Gastrointestinal (1.4%): Colitis, diarrhea, nausea, and vomiting.

Symptoms of pseudomembranous colitis can appear during or after antibiotic treatment.

Nausea and vomiting have been reported rarely.

Less frequent adverse reactions (less than 1%) are:

Cardiovascular System: Potentially life-threatening arrhythmias following rapid (less than 60 seconds) bolus administration via central venous catheter have been observed.

Hematologic System: Neutropenia, transient leukopenia, eosinophilia, thrombocytopenia and agranulocytosis have been reported. Some individuals have developed positive direct Coombs Tests during treatment with CLAFORAN and other cephalosporin antibiotics. Rare cases of hemolytic anemia have been reported.

Genitourinary System: Moniliasis, vaginitis.

Central Nervous System: Headache.

Liver: Transient elevations in SGOT, SGPT, serum LDH, and serum alkaline phosphatase levels have been reported.

Kidney: As with some other cephalosporins, interstitial nephritis and transient elevations of BUN and creatinine have been occasionally observed with CLAFORAN.

Cutaneous: As with other cephalosporins, isolated cases of erythema multiforme, Stevens-Johnson syndrome, and toxic epidermal necrolysis have been reported.

In addition to the adverse reactions listed above which have been observed in patients treated with cefotaxime sodium, the following adverse reactions and altered laboratory tests have been reported for cephalosporin class antibiotics: allergic reactions, hepatic dysfunction including cholestasis, aplastic anemia, hemorrhage, and false-positive test for urinary glucose.

Ceftriaxone Adverse Reactions

Pseudomembranous colitis has been reported with nearly all antibacterial agents, including ceftriaxone, and may range in severity from mild to life-threatening. Therefore, it is important to consider this diagnosis in patients who present with diarrhea subsequent to the administration of antibacterial agents.

Treatment with antibacterial agents alters the normal flora of the colon and may permit overgrowth of clostridia. Studies indicate that a toxin produced by Clostridium difficile is one primary cause of "antibiotic-associated colitis". After the diagnosis of pseudomembranous colitis has been established, appropriate therapeutic measures should be initiated. Mild cases of pseudomembranous colitis usually respond to drug discontinuation alone. In moderate to severe cases, consideration should be given to management with fluids and electrolytes, protein supplementation and treatment with an antibacterial drug clinically effective against Clostridium difficile colitis.

Ceftriaxone for Injection and Dextrose Injection should not be administered to hyperbilirubinemic neonates, especially prematures.LOCAL REACTIONS—Phlebitis was reported in <1% after IV administration.

HYPERSENSITIVITY—rash (1.7%). Less frequently reported (<1%) were pruritus, fever or chills.

HEMATOLOGIC—eosinophilia (6%), thrombocytosis (5.1%) and leukopenia (2.1%). Less frequently reported (<1%) were anemia, hemolytic
anemia, neutropenia, lymphopenia, thrombocytopenia and prolongation of the prothrombin time.

GASTROINTESTINAL—diarrhea (2.7%). Less frequently reported (<1%) were nausea or vomiting, and dysgeusia. The onset of
pseudomembranous colitis symptoms may occur during or after antibacterial treatment (see WARNINGS).

HEPATIC—elevations of SGOT (3.1%) or SGPT (3.3%). Less frequently reported (<1%) were elevations of alkaline phosphatase and bilirubin.

RENAL—elevations of the BUN (1.2%). Less frequently reported (<1%) were elevations ofcreatinine and the presence of casts in the urine.

CENTRAL NERVOUS SYSTEM—headache or dizziness were reported occasionally (<1%).

GENITOURINARY—moniliasis or vaginitis were reported occasionally (<1%).

MISCELLANEOUS—diaphoresis and flushing were reported occasionally (<1%).

Other rarely observed adverse reactions (<0.1%) include abdominal pain, agranulocytosis, allergic pneumonitis, anaphylaxis, basophilia, biliary lithiasis, bronchospasm, colitis, dyspepsia, epistaxis, flatulence, gallbladder sludge, glycosuria, hematuria, jaundice, leukocytosis, lymphocytosis, monocytosis, nephrolithiasis, palpitations, a decrease in the prothrombin time, renal precipitations, seizures, and serum sickness.

Canadian Adverse Reaction Newsletter, Volume 15 . Issue 1 . January 2005
Health Products and Food Branch, Marketed Health Products Directorate

Ceftriaxone (Rocephin) and immune hemolytic anemia in children

Ceftriaxone (Rocephin), marketed in Canada since Dec. 31, 1987, is a third-generation cephalosporin indicated for the treatment of susceptible strains of bacteria, as well as for prophylaxis against infections in patients undergoing hysterectomy, coronary artery bypass surgery or biliary tract surgery.1 Immune hemolytic anemia (IHA) is a hypersensitivity adverse reaction (AR) known to occur in adults and children. The Rocephin product monograph describes autoimmune hemolytic anemia as a rare AR (< 0.1% of cases),1 but does not mention IHA.

Ceftriaxone antibodies appear to be induced by an immune complex mechanism during a sensitization phase after initial exposure to the drug. 2 Intravascular hemolysis may be triggered after subsequent re-exposure. The signs and symptoms of drug-induced IHA include severe hemolytic anemia, hemoglobinuria, hypotension, acute renal failure, fever and back pain.3

From Jan. 1, 1988, to Sept. 15, 2004, Health Canada received 1 report of acute hemolysis suspected of being associated with ceftriaxone. A young child with sickle cell disease had been given a single dose of ceftriaxone (80 mg/kg body weight) intravenously for fever and cough, and within 30 minutes developed a rash, pallor and decreased level of consciousness. Laboratory examination showed a positive direct Coomb's test result, a hemoglobin level of 7 g/L (the pre-infusion level was 110 g/L) and hemolyzed red blood cells. The following day, the patient died despite resuscitation attempts. The only concomitant medication was a single oral dose of erythromycin. The patient had been exposed to ceftriaxone in the past.

Nine pediatric cases of IHA associated with exposure to ceftriaxone were identified in the literature, 6 of which were fatal.4-12One child with sickle cell anemia received ceftriaxone on several occasions and experienced 6 episodes of unexplained transient hemoglobinuria before the onset of the IHA.10

Drug-induced IHA is associated with a high mortality rate.3 Other than supportive care and red blood cell transfusion, there are few effective treatment options. Reintroduction of the drug is contraindicated because of the high risk of recurrence of hemolysis, which is often more severe.3

IHA associated with ceftriaxone is rare and has been reported to occur with repetitive, intermittent use of this drug. Children with underlying conditions such as hemoglobinopathies and immunodeficiencies are likely to require frequent treatment or prophylaxis with ceftriaxone, which may place them at increased risk of IHA. The development of signs and symptoms of IHA, including hemoglobinuria or unexplained anemia, should prompt health care professionals to consider this diagnosis and the discontinuation of the suspect drug.3

Lise Watters, MD, FRCPC; Debra Willcox, BSP, Health Canada
Rocephin (Ceftriaxone Sodium) - Contraindications, Warnings, Precautions, Adverse Reactions, Dosage and Administration Sections of Labeling Revised

BETHESDA, MD -- July 6, 2007 -- Roche and FDA informed healthcare professionals of revisions to the Contraindications, Warnings, Precautions, Adverse Reactions and Dosage and Administration sections of the prescribing information for Rocephin for Injection.

The revisions are based on new information that describes the potential risk associated with concomitant use of Rocephin with calcium or calcium containing solutions or products. Cases of fatal reactions with calcium-ceftriaxone precipitates in the lungs and kidneys in both term and premature neonates were reported. Hyperbilirubinemic neonates, especially prematures, should not be treated with Rocephin.

The drug must not be mixed or administered simultaneously with calcium-containing solutions or products, even via different infusion lines. Additionally, calcium-containing solutions or products must not be administered within 48-hours of the last administration of ceftriaxone.


1: J Infect Dis. 1989 Dec;160(6):1005-11.Links
Comment in:
J Infect Dis. 1990 Oct;162(4):991-3.
Cerebrospinal fluid endotoxin levels in children with H. influenzae meningitis before and after administration of intravenous ceftriaxone.
Arditi M, Ables L, Yogev R.

Department of Pediatrics, Children's Memorial Hospital, Northwestern University Medical School, Chicago, Illinois 60614.

Total, cell-free, and cell-bound endotoxin and bacterial density were measured in cerebrospinal fluid (CSF) of 22 children with Hemophilus influenzae meningitis. Also the effect of ceftriaxone on CSF endotoxin levels was investigated in eight patients by reexamining their CSF 2-6 h after the initial dose. Initial CSF bacterial density correlated with initial CSF endotoxin levels (P less than .001). Ceftriaxone induced a marked increase of free endotoxin in CSF, from an initial (mean +/- SE) 0.75 +/- 0.21 to 1.29 +/- 0.23 log10 ng/ml (P less than .01). This increase correlated positively with the number of bacteria killed in the CSF (P less than .01). The increase in free endotoxin was associated with an increase in mean CSF lactate levels from 8.5 to 9.7 units/l (P less than .05) and mean lactate dehydrogenase levels from 102 to 180 mmol/l (P less than .02) and a decrease in mean CSF glucose from 1.17 to 0.46 mmol/l (P less than .05). Initial CSF total endotoxin concentrations correlated both with the Herson-Todd clinical severity score (P less than .001) and with the number of febrile hospital days (P less than .001). These findings suggest that highly bactericidal agents initially lead to release of free endotoxin from gram-negative organisms into CSF, with associated enhanced inflammatory response by the host.

Different endotoxin release and IL-6 plasma levels after antibiotic administration in surgical intensive care patients
Holzheimer, RG; Hirte, JF; Reith, B; Engelhardt, W; Horak, KH; Leppert, R; Aasen, A; Capel, P; Urbaschek, R; Karch, H; Thiede, A
Journal of Endotoxin Research [J. ENDOTOXIN RES.]. Vol. 3, no. 3. 1996.

Despite the use of broad-spectrum antibiotics, aggressive fluid resuscitation, vasopressor support, the mortality associated with Gram-negative sepsis and septic shock has not decreased significantly in the last two decades. The consequences of host exposure to endotoxin and the relationship of antibiotic administration to endotoxin release have become important areas of intense interest. In vitro studies have demonstrated that there was a difference in endotoxin release between PBP-3 specific antibiotics ( beta -lactam antibiotics) and PBP-2 specific antibiotics (carbapenems). This is the first clinical report of surgical patients admitted to the surgical and anaesthesiology intensive care unit on the missing endotoxin release after imipenem treatment; however cefotaxime and ceftriaxone showed significantly more positive endotoxin tests in the plasma when compared to imipenem. Ciprofloxacin and vancomycin were intermediate in endotoxin release and tobramycin did not cause endotoxin release. There were also significant differences in endotoxin neutralizing capacity. IL-6 levels were decreased after imipenem faster than after ceftriaxone or cefotaxime; ciprofloxacin seemed to increase IL-6. Endotoxin may be harmful in patients where the immune system has been continuously challenged. Timing, dosage, or combination with other compounds as well as the effect of antibiotics on macrophages need to be tested in larger clinical trials. In this respect a consecutive study was started.

Dianne Jacobs Thompson  Est. 2007
Also (alternative medicine featuring drugless cancer treatments)
Author publication: NEXUS MAGAZINE "Seawater--A Safe Blood Plasma Substitute?"