The Shaken Baby Syndrome Myth
renamed "Abusive Head Trauma" or "Non-Accidental Injury"



* SBS began as an unproven theory and medical opinions, now discredited by biomechanical engineering studies
* No DIFFERENTIAL DIAGNOSIS done to eliminate other causes, abuse assumed without evidence
* Shaken Baby diagnostic symptoms not caused by shaking
* Child protective agencies snatch children, destroy families based on medical accusations without proof of wrong-doing
*Poor or deceptive police investigations, falsified reports, perjured testimony threaten legal rights, due process
* Prosecutors seek "victory", over justice; defense attorneys guilty of ineffective counsel, ignorance, lack of effort
* Care-takers threatened, manipulated, in order to force plea bargains, false confessions
* A fractured criminal justice system--a big piece for the rich, a small piece for the poor, and none for alleged SBS cases.



Related websites/ important people and projects ShakenBabySyndrome/Vaccines/YurkoProject
"Shaken Baby Syndrome or Vaccine Induced Encephalitis-- Are Parents Being Falsely Accused?" by Dr Harold Buttram, with Christina England (WEBSITE)
Evidence Based Medicine and Social Investigation:
EBMSI conferences, resources and information Articles and Reports
VacTruth: Jeffry Aufderheide; The SBS conection and other dangerous or deadly side effects of vaccination true, suppressed history of the smallpox vaccine fraud and other books:
Patrick Jordan
Sue Luttner, must-read articles and information on Shaken Baby Syndrome: her resources link
The Amanda Truth Project: Amanda's mother speaks out at symposium
Tonya Sadowsky


Ocular Lesions in Scurvy--Conjuctival Hemorrhage, Iritis, Other Symptoms

Ocular Lesions in Scurvy

International Journal of Dermatology - Fulltext: Volume 46(2 ...
Infantile scurvy emerged as a new disease at the end of the 19th and 20th centuries, ...
Eyelid and linear subungual hemorrhages have been described.6 ...

American Journal of Clinical Nutrition, Vol 22, 559-567, Copyright © 1969 by The American Society for Clinical Nutrition, Inc.
Ocular Lesions in Scurvy

1 From The Department of Internal Medicine, University Hospitals, Iowa City, Iowa

Despite the recorded rarity of ocular vascular lesions in human scurvy, conjunctival lesions appeared during deprivation of ascorbic acid in five of nine volunteers who participated in two studies of experimental scurvy in man, three of four men in the first study and two of five men in the second. The ocular lesions often appeared early in the development of clinical scurvy and were first noted after deprivation of ascorbic acid for 74-95 days.

These lesions varied from minute bulbar conjunctival hemorrhages and varicosities to a large subconjunctival hemorrhage accompanied by palpebral petechial hemorrhages, and conjunctival congestion.
Incidence and Significance of Hypovitaminemia in a Randomly Selected Municipal Hospital Population
CM LEEVY, L CARDI, O FRANK, R GELLENE, H BAKER - American Journal of Clinical Nutrition, 1965 - Am Soc Nutrition
... in three, varicose veins in two, dermatitis in one, iritis in one ... with special attention
to dietary history and stigmata of nutritional deficiency. ... VITAMIN C ...


Iritis is an inflammation of the iris, the protion of the eye that opens and closes to adjust the light level that passes into the eye. The light hole the iris creates is the pupil.

Like so many problems, the conventional medical community has little to offer the people that suffer from this serious, painful and chronic condition. For more information and a sufferers bulletin board, please visit

After I sent out my newsletter about "-itis" diseases, I received an e-mail from a reader that has had success treating his iritis. This is not very surprising since vitamin C is the body's chief mediator of inflammation.

EMAIL:  Submitted by: Sagarmatha2

Date: September 1, 2001

Subject: Iritis

"I have suffered from iritis at least once a year since 1989. I'm a 46-year-old male from Sweden. I've always been interested in alternative medicine and finally when my last bout came, just after Christmas last year, I came to think of using vitamin C instead of the usual cortisone prescriptions offered by the doctor.
Well, I started out by 4 g calcium ascorbate dissolved in a glass of water every morning. The pain was relieved but after a few hours I felt the pain again, so I took another 2g for every 2-3 hour during the day. Before going to bed I took a dose of 4g.

So my daily consumption was about 18-20 g/day. This costs only about 1$. After about 3 weeks I started to lower the dose gradually. And if I remember correct I was down to 10 g/day after 5 weeks. After that I have been taking 4g of C every day divided into 4 doses.

Still no bout."
Vitamin C
Function and source

Vitamin C is a water soluble vitamin ingested through
the diet in the form of citrus fruits in particular, as well
leafy green vegetables. Vitamin C is a co-enzyme that is
involved in the growth and repair of bodily tissue
through the production of collagen, as well as the
synthesis of the catecholamines adrenaline, nor-adrenaline
and dopamine. It is also an anti-oxidant, attacking
free radicals present in body tissues.

Effects of a critical deficiency

Vitamin C deficiency or scurvy presents with systemic
manifestations including bruising, bleeding gums, joint
pain and swelling, fatigue, anaemia, poor wound healing
(Hirschmann and Raugi, 1999; Olmedo et al., 2006),
and even death.

Deficient populations and causes in the developed world

In a large US survey (about 30 000 individuals), 14%
males and 10% females were reported to be deficient in
vitamin C (Hampl et al., 2004). Causes of vitamin C
deficiency include smoking, alcoholism and inadequate
dietary intake

Ocular and visual effects of a deficiency

Ocular changes result from vitamin C deficiency,
although usually only when the deficiency is severe
and prolonged. Therefore, ocular manifestations will
probably never be observed in isolation. The commonest
ocular changes include small to large subconjunctival
haemorrhages (Hood and Hodges, 1969) and orbital
haemorrhages (Sloan et al., 1999). Ocular conditions
consequent to vitamin C deficiency that are more rarely
observed include chemosis, episcleritis, keratitis, corneal
opacities, hyperaemia of the iris, iritis, anterior chamber
haemorrhage, posterior synechiae, macular exudates,
central retinal vein thrombosis, papilloedema and optic
atrophy (Hood and Hodges, 1969).


Dianne Jacobs Thompson  Est. 2007
Also (alternative medicine featuring drugless cancer treatments)
Author publication: NEXUS MAGAZINE "Seawater--A Safe Blood Plasma Substitute?"